Sudden decrease in end-tidal carbon-dioxide in a neonate undergoing surgery for type B interrupted aortic arch.

dc.contributor	Misra, Satyajeet
dc.contributor	Koshy, Thomas
dc.contributor	Mahaldar, Divya Amol Chandran
dc.date.accessioned	2012-12-04T11:45:21Z
dc.date.available	2012-12-04T11:45:21Z
dc.date.issued	2011
dc.description.abstract	Interruption of the aortic arch is a rare anomaly affecting 1% of children with congenital heart disease. The systemic circulation is ductal dependent and is determined principally by the ratio of the resistances in the systemic and the pulmonary vascular bed. Any increase in the pulmonary vascular resistance may increase the dead space ventilation due to acute pulmonary hypoperfusion. We report a case where sudden decreases in the end-tidal carbon-dioxide due to pulmonary hypoperfusion mimicked accidental endotracheal tube extubation in an infant undergoing repair of interrupted aortic arch.
dc.identifier.citation	Annals of cardiac anaesthesia. 14; 3; 206-10	en_US
dc.identifier.uri	https://dspace.sctimst.ac.in/handle/123456789/1110
dc.publisher	Annals of cardiac anaesthesia
dc.subject	Critical Care
dc.title	Sudden decrease in end-tidal carbon-dioxide in a neonate undergoing surgery for type B interrupted aortic arch.