PROBING THE HEART OF CARDIOMYOPATHIES

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dc.contributor.authorKARTHA, CC
dc.contributor.authorSHIVAKUMAR, K
dc.date.accessioned2017-03-10T03:28:16Z
dc.date.available2017-03-10T03:28:16Z
dc.date.issued1993
dc.description.abstractCurrent knowledge of aetiopathogenesis of various forms of human cardiomyopathy is reviewed in this article. There are overwhelming evidences to suggest a pathogenetic link between persistent viral infection, induction of autoimmunity (influenced by immune response determinants in the HLA region) and alteration in the differentiated functions of the myocardium in dilated cardiomyopathy. Genetic studies have identified specific defects in patients with hypertrophic cardiomyopathy but the cellular basis of the disease remains unresolved. Endomyocardial diseases associated with hypereosinophilic syndrome and endomyocardial fibrosis of the tropics appear to be two separate entities. A unifying link among endomyocardial fibrosis of diverse causes could be the uniform response of the 'cardiac tissue to injuries of heterogenous nature. Application of microtechniques to endomyocardial biopsy promises definitive information on aetiopathogenesis of cardiomyopathies.
dc.identifier.citation65 ,6;455-461en_US
dc.identifier.urihttps://dspace.sctimst.ac.in/handle/123456789/10310
dc.publisherCURRENT SCIENCE
dc.subjectScience & Technology - Other Topics
dc.titlePROBING THE HEART OF CARDIOMYOPATHIES
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